References
[1] B. Ghosh, M.
Mukhopadhyay, and D. Bhattacharya, “Biopolymer-based nanofilms for the
treatment of burn injuries,” Biopolym. Nano Film. Appl. Food Packag. Injury
Heal., pp. 311–336, Jan. 2021, doi: 10.1016/B978-0-12-823381-8.00005-3.
[2] “Burns: Types,
Symptoms, and Treatments.”
[3] J. L. Hunt, B. D.
Arnoldo, and G. F. Purdue, “Chapter 4 - Prevention of burn injuries,” D. N. B.
T.-T. B. C. (Fourth E. Herndon, Ed. London: W.B. Saunders, 2012, pp. 47-55.e2.
doi: https://doi.org/10.1016/B978-1-4377-2786-9.00004-7.
[4] “The global
burden of disease : 2004 update.”
[5] “Institute for
Health Metrics and Evaluation |.”
[6] “Burns | National
Health Portal Of India.”
[7] J. W. L. Davies,
“The problems of burns in India,” Burns, vol. Suppl 1, no. SUPPL. 1,
1990.
[8] L. K. Branski, D.
N. Herndon, and R. E. Barrow, “Chapter 1 - A brief history of acute burn care
management,” D. N. B. T.-T. B. C. (Fourth E. Herndon, Ed. London: W.B.
Saunders, 2012, pp. 1-7.e2. doi:
https://doi.org/10.1016/B978-1-4377-2786-9.00001-1.
[9] M. D. Peck,
“Epidemiology of burns throughout the world. Part I: Distribution and risk
factors,” Burns, vol. 37, no. 7, pp. 1087–100, Nov. 2011, doi:
10.1016/j.burns.2011.06.005.
[10] K. R. Kasten, A.
T. Makley, and R. J. Kagan, “Update on the critical care management of severe
burns,” J. Intensive Care Med., vol. 26, no. 4, pp. 223–236, 2011, doi:
10.1177/0885066610390869.
[11] S. Taylor, J.
Jeng, J. R. Saffle, S. Sen, D. G. Greenhalgh, and T. L. Palmieri, “Redefining
the outcomes to resources ratio for burn patient triage in a mass casualty,” J.
Burn Care Res., vol. 35, no. 1, pp. 41–45, 2014, doi:
10.1097/BCR.0000000000000034.
[12] S. V. Mahadevan
and G. M. Garmel, An introduction to clinical emergency medicine, 2nd
ed. Cambridge: Cambridge University Press, 2012.
[13] M. Jeschke, Handbook
of Burns Volume 1: Acute Burn Care. Springer, 2012.
[14] M. Peden, World
report on child injury prevention. Geneva, Switzerland: World Health
Organization, 2008.
[15] S. Eisen and C.
Murphy, Training in paediatrics : the essential curriculum. Oxford:
Oxford University Press, 2009.
[16] R. K. Jutla and D.
Heimbach, “Love burns: An essay about bride burning in India,” J. Burn Care
Rehabil., vol. 25, no. 2, pp. 165–70, Mar. 2004, doi:
10.1097/01.bcr.0000111929.70876.1f.
[17] J. A. Haagsma et
al., “The global burden of injury: incidence, mortality,
disability-adjusted life years and time trends from the Global Burden of
Disease study 2013,” Inj. Prev., vol. 22, no. 1, pp. 3–18, Feb. 2016,
doi: 10.1136/INJURYPREV-2015-041616.
[18] K. S. Wood and P.
V Gordon, “Neonatal and Pediatric Transport,” Tintinalli’s Emerg. Med. A
Compr. Study Guid., pp. 16–21, 2011.
[19] G. Arturson,
“Pathophysiology of the burn injury.,” Ann. Chir. Gynaecol., vol. 69,
no. 5, pp. 178–190, 1980.
[20] “Weissman1990
Estado Del Arte 1.”
[21] Z. Xing et al.,
“IL-6 is an antiinflammatory cytokine required for controlling local or
systemic acute inflammatory responses,” J. Clin. Invest., vol. 101, no.
2, pp. 311–320, 1998, doi: 10.1172/JCI1368.
[22] W. L. Biffl, E. E.
Moore, F. A. Moore, and V. M. Peterson, “Interleukin-6 in the injured patient.
Marker of injury or mediator of
inflammation?,” Ann. Surg., vol. 224, no. 5, pp. 647–664, Nov.
1996, doi: 10.1097/00000658-199611000-00009.
[23] A. Gosain and R.
L. Gamelli, “A primer in cytokines.,” J. Burn Care Rehabil., vol. 26,
no. 1, pp. 7–12, 2005, doi: 10.1097/01.bcr.0000150214.72984.44.
[24] G. D. Kay,
“Prolonged Survival of a Skin Homograft in a Patient With Very Extensive
Burns,” Ann. N. Y. Acad. Sci., vol. 64, no. 5, pp. 767–774, 1957, doi:
10.1111/j.1749-6632.1957.tb52471.x.
[25] L. F. Rose and R.
K. Chan, “The Burn Injury Microenvironment.,” Adv. injury care, vol. 5,
no. 3, pp. 106–118, Mar. 2016, doi: 10.1089/injury.2014.0536.
[26] V. Suvatte, C.
Chuntrasakul, M. Tuchinda, N. Srimaruta, and A. Assateerawatts,
“8ImmunologicalIncompetenc-inBurnPatientsAPJAIVol1No2DecP112.pdf.”
[27] J. B. Grogan,
“Altered neutrophil phagocytic function in burn patients,” J. Trauma,
vol. 16, no. 9, pp. 734–738, 1976, doi: 10.1097/00005373-197609000-00009.
[28] A. Göebel et
al., “Injury induces deficient interleukin-12 production, but
interleukin-12 therapy after injury
restores resistance to infection.,” Ann. Surg., vol. 231, no. 2, pp.
253–261, Feb. 2000, doi: 10.1097/00000658-200002000-00015.
[29] R. N. Stephan, A.
Ayala, J. M. Harkema, R. E. Dean, J. R. Border, and I. H. Chaudry, “Mechanism
of immunosuppression following hemorrhage: defective antigen presentation by macrophages.,” J. Surg.
Res., vol. 46, no. 6, pp. 553–556, Jun. 1989, doi:
10.1016/0022-4804(89)90019-x.
[30] O. Loughlin and L.
S. Sorell, “United States Patent [ 191 Date of Patent :,” vol. 2, no. 12, 1988.
[31] D. G. Burleson, A.
D. J. Mason, and B. A. J. Pruitt, “Lymphoid subpopulation changes after thermal
injury and thermal injury with infection
in an experimental model.,” Ann. Surg., vol. 207, no. 2, pp. 208–212,
Feb. 1988, doi: 10.1097/00000658-198802000-00016.
[32] B. Muthiah, F. K.
Ofori-kuma, C. B. Rao, K. Mealy, and J. B. O’Mahony, “A review of a five-year
experience with the open mesh repair for inguinal hernia,” Ir. J. Med. Sci.,
vol. 171, no. 1, p. 24, 2002, doi: 10.1007/BF03170369.
[33] J. H. N. Wolfe, A.
V. O. Wu, I. Saporoschetz, J. A. Mannick, and N. E. O. Connor, “and Impaired
Lymphocyte Blastogenesis in Burn Patients,” 2015.
[34] P. Casson, A. C.
Solowey, J. M. Converse, and F. T. Rapaport, “Delayed hypersensitivity status
of burned patients.,” Surg. Forum, vol. 17, pp. 268–270, 1966.
[35] M. G. Dehne, A.
Sablotzki, A. Hoffmann, J. Mühling, F. E. Dietrich, and G. Hempelmann,
“Alterations of acute phase reaction and cytokine production in patients
following severe burn injury.,” Burns,
vol. 28, no. 6, pp. 535–542, Sep. 2002, doi: 10.1016/s0305-4179(02)00050-5.
[36] D. Church, S.
Elsayed, O. Reid, B. Winston, and R. Lindsay, “Burn injury infections.,” Clin.
Microbiol. Rev., vol. 19, no. 2, pp. 403–434, Apr. 2006, doi:
10.1128/CMR.19.2.403-434.2006.
[37] “Burn
Complications | Burn Injury Guide.”
[38] “Microbial
Infection of Burn Injuries - microbewiki.”
[39] N. S. Gibran and
D. M. Heimbach, “Current status of burn injury pathophysiology.,” Clin.
Plast. Surg., vol. 27, no. 1, pp. 11–22, Jan. 2000.
[40] L. T. Vo, G. D.
Papworth, P. M. Delaney, D. H. Barkla, and R. G. King, “A study of vascular
response to thermal injury on hairless mice by fibre optic confocal imaging,
laser doppler flowmetry and conventional histology,” Burns, vol. 24, no.
4, pp. 319–324, 1998, doi: 10.1016/S0305-4179(98)00028-X.
[41] D. L. Steed, “The
role of growth factors in injury healing.,” Surg. Clin. North Am., vol.
77, no. 3, pp. 575–586, Jun. 1997, doi: 10.1016/s0039-6109(05)70569-7.
[42] “madden1971.pdf.”
[43] R. Gomez et al.,
“Causes of mortality by autopsy findings of combat casualties and civilian patients admitted to a burn unit.,” J. Am.
Coll. Surg., vol. 208, no. 3, pp. 348–354, Mar. 2009, doi:
10.1016/j.jamcollsurg.2008.11.012.
[44] E. F. Keen et
al., “Incidence and bacteriology of burn infections at a military burn
center,” Burns, vol. 36, no. 4, pp. 461–468, 2010, doi:
10.1016/j.burns.2009.10.012.
[45] M. A. Albrecht et
al., “Impact of Acinetobacter Infection on the Mortality of Burn Patients,”
J. Am. Coll. Surg., vol. 203, no. 4, pp. 546–550, 2006, doi:
10.1016/j.jamcollsurg.2006.06.013.
[46] E. E. Horvath et
al., “Fungal injury infection (not colonization) is independently
associated with mortality in burn
patients.,” Ann. Surg., vol. 245, no. 6, pp. 978–985, Jun. 2007, doi:
10.1097/01.sla.0000256914.16754.80.
[47] S. Sarabahi, V. K.
Tiwari, S. Arora, M. R. Capoor, and A. Pandey, “Changing pattern of fungal
infection in burn patients,” Burns, vol. 38, no. 4, pp. 520–528, 2012,
doi: 10.1016/j.burns.2011.09.013.